Among other things, these two anti-estrogens will continue to foster LH release as testosterone levels start ~ go back up, as well as combat any potential estrogenic side effects that may be caused by HCG’s up-regulation < I testicular aromatase 50 Although in the first couple of weeks the anti-estrogens probably do very little, they should be much more helpful towards the middle and end of the program. During this clinical investigation: normal hormonal function was restored in all subjects I within 45 days of drug cessation. This is a definite success far more favorable than the protracted recovery window noted in studies without post-cycle therapy, such as the 250 mg/week testosterone enanthate investigation, highlighted in Figure I. For me, I believe such a detailed recovery program should follow any serious steroid cycle it is the best way to maintain your gains at their maximum and that is, after all, what we are after.
HCG within the medical field is primarily administered via intramuscular (IM) injections, although it can also be administered subcutaneously, which has also become just as frequent as IM injections. Studies have found that when intramuscular and subcutaneous injections of HCG were compared, the results were almost the exact same for both, indicating almost no difference between the two  . The only difference between the two methods of injection is the difference in the rate of release from the injection site and the time required for peak blood plasma levels to be reached (6 hours for IM, and 16 – 20 hours for subcutaneous). The majority of anabolic steroid users will elect to inject HCG subcutaneously.
These are not very high levels and I was surprised that I felt so good at that low of a level. And this brings up a major theory of mine: the more you (naturally) boost your nitric oxide and lower inflammation, the less testosterone that you need. Testosterone activates eNOS, the enzyme that controls your endothelial nitric oxide, which is your primary engine for NO. So I always felt that men with arterial issues probably subconsciously pushed for higher T levels to compensate. And I have been working very hard on my arteries and feel this may be part of the reason I have been able to live with lower testosterone levels.